WebMay 2, 2008 · The p38 mitogen-activated protein kinase (MAPK) is activated through phosphorylation primarily by MAPK kinase 3 (MKK3) and MKK6 in response to cellular stress and cytokines. The p38 MAPK pathway functions in the control of differentiation, the blockade of proliferation, and in the induction of apoptosis ( 1 ). WebJan 3, 2024 · Exploring the p38-MAPK Signaling Pathway eu_abclonal Leave a Comment on Exploring the p38-MAPK Signaling Pathway When I was an aspiring (much younger) scientist, one of the challenges was finding quality antibodies to accommodate our research group’s high-throughput Western blotting platform 1 while studying signaling pathways in …
Anti-p38 alpha/MAPK14 (phospho Y322) antibody …
WebMonoclonal Antibody for studying ERK1 (Thr202/Tyr204) phosphate/ERK2 (Thr185/Tyr187) phosphate. Cited in 950 publications. Validated for WB, IP, IHC. Highly specific and rigorously validated in-house, Phospho-p44/42 MAPK (Erk1/2) (Thr202/Tyr204) (20G11) Rabbit Monoclonal Antibody (CST #4376) is ready to ship. WebNov 29, 2006 · p38 MAPK is a member of the mitogen-activated protein kinases (MAPKs) that mediate inflammation, cell proliferation, and apoptosis ( Seger and Krebs, 1995; Cobb, 1999 ), and can be activated by the adenosine A 1 receptor via a G-protein-dependent pathway ( Robinson and Dickenson, 2001 ). エレベーター屋 転職
Anti-Phospho-p38 MAPK-T180/Y182 antibody produced in rabbit
WebJan 5, 2016 · The panels on the left and the panels on the right are from the same Western blot, but from different regions of the SDS-PAGE gel. (B) Quantification of the ratio of anti-phospho-p38 MAPK antibody signal to anti-actin antibody signal, normalized to the value of wild-type L4 animals. Values indicate an average from three independent Western blots. WebRabbit recombinant monoclonal p38 (phospho T180 + Y182) antibody [P38T180Y182-E3] conjugated to APC. Validated in Flow Cyt and tested in Rat. Immunogen corresponding to synthetic peptide. WebApr 14, 2024 · Moreover, p-p38 MAPK-positive cells were colocalized with increased Iba-1 in the TNC. Our findings indicate that rapamycin treatment reduces both mechanical and cold orofacial allodynia in mice with trigeminal neuropathic pain, which is closely associated with the modulation of p-MKK4/p-p38 MAPK-mediated microglial activation in the TNC. エレベーター 庫